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dc.contributor.authorStäudle, Benjamin
dc.contributor.authorSeynnes, Olivier R.
dc.contributor.authorLaps, Guido
dc.contributor.authorGöll, Fabian
dc.contributor.authorBrüggemann, Gert-Peter
dc.contributor.authorAlbracht, Kirsten
dc.date.accessioned2022-07-26T10:35:51Z
dc.date.available2022-07-26T10:35:51Z
dc.date.created2021-08-06T11:19:24Z
dc.date.issued2021
dc.identifier.citationMedicine & Science in Sports & Exercise. 2021, 53(7), 1356-1366en_US
dc.identifier.issn0195-9131
dc.identifier.urihttps://hdl.handle.net/11250/3008553
dc.descriptionDette er siste tekst-versjon av artikkelen, og den kan inneholde små forskjeller fra forlagets pdf-versjon. Forlagets pdf-versjon finner du på lww.com / This is the final text version of the article, and it may contain minor differences from the journal's pdf version. The original publication is available at lww.comen_US
dc.description.abstractIntroduction: Achilles tendon rupture (ATR) patients have persistent functional deficits in the triceps surae muscle–tendon unit (MTU). The complex remodeling of the MTU accompanying these deficits remains poorly understood. The purpose of the present study was to associate in vivo and in silico data to investigate the relations between changes in MTU properties and strength deficits in ATR patients. Methods: Eleven male subjects who had undergone surgical repair of complete unilateral ATR were examined 4.6 ± 2.0 (mean ± SD) yr after rupture. Gastrocnemius medialis (GM) tendon stiffness, morphology, and muscle architecture were determined using ultrasonography. The force–length relation of the plantar flexor muscles was assessed at five ankle joint angles. In addition, simulations (OpenSim) of the GM MTU force–length properties were performed with various iterations of MTU properties found between the unaffected and the affected side. Results: The affected side of the patients displayed a longer, larger, and stiffer GM tendon (13% ± 10%, 105% ± 28%, and 54% ± 24%, respectively) compared with the unaffected side. The GM muscle fascicles of the affected side were shorter (32% ± 12%) and with greater pennation angles (31% ± 26%). A mean deficit in plantarflexion moment of 31% ± 10% was measured. Simulations indicate that pairing an intact muscle with a longer tendon shifts the optimal angular range of peak force outside physiological angular ranges, whereas the shorter muscle fascicles and tendon stiffening seen in the affected side decrease this shift, albeit incompletely. Conclusions: These results suggest that the substantial changes in MTU properties found in ATR patients may partly result from compensatory remodeling, although this process appears insufficient to fully restore muscle function.en_US
dc.language.isoengen_US
dc.subjecttendon ruptureen_US
dc.subjectstiffnessen_US
dc.subjectsimulationen_US
dc.subjectmuscle forceen_US
dc.subjectmuscle fascicleen_US
dc.subjecttendon healingen_US
dc.titleRecovery from Achilles Tendon Repair: A Combination of Postsurgery Outcomes and Insufficient Remodeling of Muscle and Tendonen_US
dc.title.alternativeRecovery from Achilles Tendon Repair: A Combination of Postsurgery Outcomes and Insufficient Remodeling of Muscle and Tendonen_US
dc.typePeer revieweden_US
dc.typeJournal articleen_US
dc.description.versionacceptedVersionen_US
dc.source.pagenumber1356-1366en_US
dc.source.volume53en_US
dc.source.journalMedicine & Science in Sports & Exerciseen_US
dc.source.issue7en_US
dc.identifier.doi10.1249/MSS.0000000000002592
dc.identifier.cristin1924348
dc.description.localcodeInstitutt for fysisk prestasjonsevne / Department of Physical Performanceen_US
cristin.ispublishedtrue
cristin.fulltextpostprint
cristin.qualitycode2


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